Interaksi Faktor Genetik dan Lingkungan pada Attention Deficit/Hyperactivity Disorder (ADHD)
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Attention Deficit /Hyperactivity Disorder (ADHD) merupakan gangguan neurodevelopmental yang ditandai dengan kesulitan memusatkan perhatian disertai hiperaktivitas dan impulsivitas, yang terjadi . Prevalensi global cukup tinggi diantara anak-anak, dan lebih dari setengahnya berlanjut hingga dewasa. Faktor genetik memegang peranan penting pada ADHD. Polimorfisme sejumlah gen ditengarai berperan pada sintesa molekuler bermasalah pada ADHD. Faktor lingkungan juga berhubungan dengan risiko ADHD. Paparan faktor lingkungan dapat memoderasi faktor genetik pada ADHD, dan demikian juga faktor genetik menyumbang kerentanan pada individu ADHD saat terpapar faktor risiko dari lingkungan. Interaksi faktor genetik dan lingkungan juga berkontribusi terhadap risiko ADHD, meski mekanisme interaksi kedua faktor ini belum diketahui secara pasti.
Andrés Martin, E. et al. (2018) LEWIS'S CHILD AND ADOLESCENT PSYCHIATRY A Comprehensive Textbook FIFTH EDITION.
APA (2013) DSM-5. 5th edn. Washington DC: American Psycjiatric Publishing.
Cortese, S. and Coghill, D. (2018) ‘Twenty years of research on attention-deficit / hyperactivity disorder (ADHD): looking back, looking forward', Evidence Based Mental Health, pp. 1–4.
Daneshparvar, M. et al. (2016) ‘The Role of Lead Exposure on Attention-Deficit/ Hyperactivity Disorder "Žin Children: A Systematic Review.', Iranian journal of psychiatry, 11(1), pp. 1–14.
Dark, C., Homman-ludiye, J. and Bryson-richardson, R. J. (2018) ‘The Role of ADHD Associated Genes in Neurodevelopmen', Developmental Biology. Elsevier Inc.
Van Doren, J. et al. (2018) ‘Sustained effects of neurofeedback in ADHD: a systematic review and meta-analysis', European Child and Adolescent Psychiatry. Springer Berlin Heidelberg, (0123456789), pp. 1–13
Faraone, S. V. and Larsson, H. (2018) ‘Genetics of attention deficit hyperactivity disorder', Molecular Psychiatry, pp. 1–14.
Franke, B. et al. (2017) ‘Live fast, die young? A review on the developmental trajectories of ADHD across the lifespan Article', Submitted. Elsevier B.V., pp. 1–30. doi: 10.1016/j.euroneuro.2018.08.001.
Froehlich, T. E. et al. (2011) ‘Update on Environmental Risk Factors for Attention-Deficit / Hyperactivity Disorder', pp. 333–344.
Geier, D. A. et al. (2018) ‘A cross-sectional study of the relationship between infant Thimerosal-containing hepatitis B vaccine exposure and attention-deficit/hyperactivity disorder', Journal of Trace Elements in Medicine and Biology. Elsevier, 46(October 2017), pp. 1–9.
Gould, K. L. et al. (2018) ‘Gene-Environment Interactions in ADHD: The Roles of SES and Chaos', Journal of Abnormal Child Psychology. Journal of Abnormal Child Psychology, 46(2), pp. 251–263.
Hong, S.-B. (2015) ‘Environmental Lead Exposure and Attention Deficit / Hyperactivity Disorder Symptom Domains in a Community Sample of South Korean School-Age Children', Environmental Health Perspectives, 123(3), pp. 271–276.
Li, Z. et al. (2014) ‘Molecular genetic studies of ADHD and its candidate genes: A review', Psychiatry Research. Elsevier, 219(1), pp. 10–24
Lir-wan, F. and Yi, P. (2017) ‘Dysregulation of neurogenesis by neuroinflammation: key differences in neurodevelopmental and neurological disorders'.
Man, K. K. C. et al. (2018) ‘Prenatal antidepressant exposure and the risk of attention-deficit hyperactivity disorder in children: A systematic review and meta-analysis', Neuroscience and Biobehavioral Reviews, 86(December 2017), pp. 1–11.
Maric, M. and Bögels, S. M. (2018) ‘Parenting behaviors associated with youth AD diagnosis vs. youth ADHD diagnosis', European Journal of Psychiatry. Asociación Universitaria de Zaragoza para el Progreso de la Psiquiatría y la Salud Mental, (xx).
Nigg, J., Nikolas, M. and Burt, S. A. (2010) ‘Measured gene-by-environment interaction in relation to attention-deficit/hyperactivity disorder', Journal of the American Academy of Child and Adolescent Psychiatry. Elsevier Inc., 49(9), pp. 863–873.
Nigg, J. T. et al. (2012) ‘Meta-Analysis of Attention-Deficit/Hyperactivity Disorder or Attention-Deficit/Hyperactivity Disorder Symptoms, Restriction Diet, and Synthetic Food Color Additives', JAAC. Elsevier Inc., 51(1), p. 86–97.e8.
Perera, F. P. et al. (2018) ‘Combined effects of prenatal exposure to polycyclic aromatic hydrocarbons and material hardship on child ADHD behavior problems', Environmental Research. Elsevier Inc., 160(September), pp. 506–513. doi: 10.1016/j.envres.2017.09.002.
Sadock, B. J., Sadock, V. A. and Ruiz, P. (2015) Synopsis of Psychiatry: Behavioral Science/ Clinical Psychiatry, Synopsis of Psychiatry: Behavioral Science/ Clinical Psychiatry 11Th Edition.
Saha, T. et al. (2018) ‘Genetic variants of the folate metabolic system and mild hyperhomocysteinemia may affect ADHD associated behavioral problems', Progress in Neuro-Psychopharmacology and Biological Psychiatry. Elsevier, 84(September 2017), pp. 1–10.
Sayal, K. et al. (2018) ‘ADHD in children and young people: prevalence, care pathways, and service provision', The Lancet Psychiatry. Elsevier Ltd, 5(2), pp. 175–186.
Schneider-Momm, K. et al. (2018) ‘Food intolerance and ADHD', Neurology Psychiatry and Brain Research. Elsevier GmbH., 29, p. 22.
Sprich, S. E. et al. (2016) ‘Cognitive-Behavioral Therapy for ADHD in Adolescents: Clinical Considerations and a Case Series', Cognitive and Behavioral Practice, 22(2), pp. 116–126.
Stahl, S. M. (2013) Stahl's Essential Psychopharmacology 4th Edition. 4th edn. new york, USA: Cambridge University Press.
Waldie, K. E. et al. (2017) ‘Dopamine transporter (DAT1/SLC6A3) polymorphism and the association between being born small for gestational age and symptoms of ADHD', Behavioural Brain Research. Elsevier, 333(June), pp. 90–97
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